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What you don’t know can kill you and the people you love!

ken russell
user 9323823
Austin, TX
Post #: 3
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What you don’t know can kill you and the people you love!

Ethnic Nigerian babies born in Sweden have a 20,000% increase in the probability of being autistic when compared to Swedish babies.

African Americans have a 2000% higher early cardiac mortality rate (between the ages of 40 and 50) when compared to White Americans.

90% of African American high school girls (who are not athletes) in Milwaukee, Wisconsin already have bone density issues.

The examples listed above highlight the tragic significance of vitamin d3 deficiency in the health of people of color who live in northern latitudes were UVB radiation from the sun is minimized.

We are what we eat!

Poor women in Japan (who eat almost no meat or dairy) have been diagnosed with only 10% of the breast cancer rates of Americans for the last 60 years. Rich women in Japan (who have eaten meat and dairy daily) have an 850% higher incidence of breast cancer when compared to poor Japanese women who eat little meat or dairy.

30% of Hispanic American children are now pre-diabetic at age 12.

40% of Americans between the ages of 40 and 70 are pre-diabetic.

Diabetics at age 45 will have 500% more cancer, 1400% more heart attacks and 3000% more strokes when compared with non-diabetics.
The U.S. Surgeon General and the World Health Organization have stated that between 60% and 70% of heart disease, cancer, diabetes and strokes are preventable with changes in diet.

We now know how to use minor changes in diet, supplementation and exercise to pursue to the 70% reduction referenced above.

A new T2 Diabetes intervention process based on 30 year old science optimizes the bioavailability of chromium III ions in the tyrosine kinase insulin receptor activity. The downstream benefits of this intervention may have a profound influence on more than fifty different metabolic disorders where elevated insulin and glucose are present. We have received testimonials of profound symptomatic relief from T2 Diabetes, Restless Legs Syndrome (RLS) and Wasting Muscle Disease (Sarcopenia). Lower ambient insulin levels will likely have a positive impact on PCOS and endometriosis. Increased glucose uptake into the heart muscle will likely postpone the fatty acid substrate shift in the later progression of ischemic heart disease. Lower HbA1c numbers make it possible for the brain to upload more tryptophan for serotonin conversion and the reduction of nicotine cravings. Immediate counter regulatory response to lower glucose numbers includes secretion of human growth hormone and insulin growth factor 1 (for enhanced bone metabolism, enhanced fatty acid oxidation and enhanced protein synthesis of ligaments, tendons and connective tissue).

This alternative T2 Diabetes treatment process actually addresses the underlying causation of T2 Diabetes instead of simply masking over the primary symptom of high glucose levels. One of the most important elements is the ability to temporarily suspend the condition defined as "Insulin Resistance" and facilitate rapid induction of as much as 70% of the total body serum glucose into the skeletal muscles of an unmedicated T2 Diabetic, in less than an hour..

In vivo studies document serum glucose reductions as much as 238 mg/dL down to 65 mg/dL in less than an hour. Because the muscle cells are temporarily satiated with glucose, the glucagon-signaling action is dramatically reduced, the liver reduces the rapid release of glucose into the blood, and lower overnight glucose levels become much more obtainable. When these methods are utilized before bedtime, it is possible for T2 Diabetics to:

(1) retire for the evening with low glucose numbers (55 - 70mg/dl),
(2) restore normal sleep patterns, lipid profiles and human growth hormone secretions,
(3) reduce depression and carbohydrate craving,
(4) increase muscle mass, brain function, energy levels & weight loss.

This rapid glucose reduction process is temporary and must be repeated daily. One application should be planned to coincide with the post prandial glucose spike after the last meal of the day. This increases the likelihood of quality sleep with a lower serum glucose profile. Another application could be planned to coincide with the post prandial glucose spike after the first meal of the day. This would enhance of the benefits of food satiation and provide the energy requirements for the much of that day. Increased brain serotonin levels and of reduced anxiety will tend to follow from each application.

Alzheimer's disease has recently been identified as an insulin mediated glucose uptake failure into brain neurons. Retinitis pigmentosa has recently been identified as an insulin mediated glucose uptake failure into rod cells. Restless legs syndrome has recently been identified as an insulin mediated iron uptake failure into the substantia nigra pars compacta of the brain. Sarcopenia has recently been identified as an insulin mediated amino acid uptake failure into muscle cells. The common element is insulin mediated uptake failure. This process solves the riddle of one facet of insulin mediated uptake failure in T2 Diabetes, RLS and Sarcopenia . It may do so in other metabolic disorders as well.

If you find this interesting, we should talk.

Ken Russell
Diabetes Prevention Project
832-655-6520
























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